AAOS Spine Surgery MCQs (Set 2): Degenerative Cervical, Lumbar Disc, & Trauma | ABOS Review

Odontoid fractures can be classified based on the anatomic position of the fracture within the dens itself. Type I is an oblique fracture through the upper part of the odontoid process. Type II is a fracture that occurs at the base of the odontoid as it attaches to the body of C2; type III occurs when the fracture line extends through the body of the axis. Type 1 fractures typically can be treated nonsurgically with 6 to 8 weeks of immobilization with a semi-rigid cervical orthosis. Nondisplaced, deep type III fractures generally are treated with skeletal halo fixation. Deep, displaced, and angled type III fractures can be treated with closed reduction and skeletal halo fixation. Shallow type III fractures are sometimes amenable to anterior odontoid screw fixation. Type II fractures can be managed nonsurgically or surgically. Treatment options include halo immobilization, internal fixation (odontoid screw fixation), and posterior atlantoaxial arthrodesis. Management with the halo vest usually is considered if the initial dens displacement is less than 6 mm, the reduction is performed within 1 week of the injury and is able to be maintained, and the patient is younger than age 60 years. Halo vest immobilization can lead to a healing rate of more than 90%. Posterior surgical fusion techniques provide high fusion success rates but do so at the expense of cervical rotation. Up to 50% of rotation is lost with these techniques. Anterior odontoid single screw fixation is often tolerated better than skeletal halo fixation and also is noted to preserve the normal rotation at C1/C2. Studies have shown less of a malunion and nonunion rate in the treatment of type II odontoid fractures with anterior odontoid screw fixation. Osteoporosis, short neck and barrel-chested anatomy, and fractures that are more than 4 weeks old preclude anterior odontoid fixation. Shilpakar S, McLaughlin MR, Haid RW Jr, et al: Management of acute odontoid fractures: Operative techniques and complication avoidance. Neurosurg Focus 2000;8:e3. Subach BR, Morone MA, Haid RW Jr, et al: Management of acute odontoid fractures with single-screw anterior fixation. Neurosurgery 1999;45:812-819.

The carotid tubercle is usually located at the level of C6. The angle of the mandible is at C1-C2; the hyoid is at C4; the superior portion of the thyroid cartilage is C4-C5; and the cricoid cartilage is at C6. Smith GW, Robinson RA: The treatment of certain cervical-spine disorders by anterior removal of the intervertebral disc and interbody fusion. J Bone Joint Surg Am 1958;40:607.

The postoperative incidence of C5 root palsy after laminoplasty ranges from 5% to 12%. Other roots also may be affected. The palsies tend to be motor dominant, although sensory dysfunction and radicular pain are also possible. The palsy may arise during the immediate postoperative period or up to 20 days later. C5 may be preferentially involved because it is at the apex of the cervical lordosis. Recovery usually occurs over weeks to months. Spivak JM, Connolly PJ (eds): Orthopaedic Knowledge Update: Spine 3. Rosemont, IL, American Academy of Orthopaedic Surgeons, 2006, pp 235-249.

Revision anterior exposure within 2 weeks of total disk replacement incurs relatively little additional morbidity because adhesion formation is minimal. Surgeons should have a low threshold for revising implants that are clearly dangerously malpositioned or show early migration within this 2-week window. Beyond this time period, a revision strategy must be individualized to the particular clinical situation. A posterior fusion with instrumentation with or without a laminectomy is currently the most effective salvage procedure.

Kramer and associates conducted a retrospective review during an "epidemic" period to identify the risk factors associated with a sudden increase in the rate of surgical site infections. They found in a multivariate analysis that the use of morphine nerve paste resulted in a 7.6-fold increase in postoperative surgical wound debridement, and an 11% rate of surgical site complications. Kramer MH, Mangram AJ, Pearson ML, et al: Surgical-site complications associated with a morphine nerve paste used for postoperative pain control after laminectomy. Infect Control Hosp Epidemiol 1999;20:183-186.

The arrow is pointing to a cystic-appearing structure with high signal intensity on T2-weighted image sequencing. It appears to be contiguous with the hypertrophied right facet joint, which appears to also have high signal intensity. The mass significantly narrows the right lateral recess. The high signal intensity suggests that this is a fluid-filled mass. In addition, the facet joints are degenerative and there is a very mild degree of anterolisthesis on the sagittal image. These findings make a lumbar synovial cyst the most likely diagnosis. Most lumbar juxtafacet cysts are observed at the L4-5 level, extradurally and adjacent to the degenerative facet joint. They may contain synovial fluid and/or extruded synovium. Presentation is indistinguishable from that of a herniated disk. The etiology of spinal cysts remains unclear, but there appears to be a strong association between their formation and worsening spinal instability. They occasionally regress spontaneously and may respond to aspiration and injection of corticosteroids, though there is a high recurrence rate with nonsurgical management. Synovial cysts resistant to nonsurgical management should be treated surgically. If the patient's symptoms can be attributable to radicular findings, a microsurgical decompression that limits further destabilization should suffice. However, if there is significant low back pain attributable to spinal instability, decompression and fusion remains an appropriate option. Banning CS, Thorell WE, Leibrock LG: Patient outcome after resection of lumbar juxtafacet cysts. Spine 2001;26:969-972. Deinsberger R, Kinn E, Ungersbock K: Microsurgical treatment of juxta facet cysts of the lumbar spine. J Spinal Disord Tech 2006;19:155-160.

The patient's history and physical examination findings are consistent with a lumbar disk herniation at the L4-5 level. Weakness of the extensor hallucis longus and gluteus medius are consistent with an L5 lumbar radiculopathy. Nerve root tension signs are also consistent with sciatica from a lumbar disk herniation. The MRI scans confirm a posterolateral disk herniation at L4-5, which typically affects the exiting L5 nerve root. Hoppenfeld S: Orthopedic Neurology. Philadelphia, PA, JB Lippincott, 1977, pp 45-74.

The CT scans reveal an occipital-cervical dissociation with subluxation of the occipitocervical joints bilaterally. Definitive management should consist of an occipital-cervical fusion with instrumentation. Immobilization in a Philadelphia collar is inadequate for this highly unstable injury, and halo immobilization, while affording adequate temporary immobilization, is not appropriate definitive management for this ligamentous injury. The patient does not have an injury at C4-C5 or C6-C7. Jackson RS, Banit DM, Rhyne AL III, et al: Upper cervical spine injuries. J Am Acad Orthop Surg 2002;10:271-280.

The patient has a burst fracture with probable unilateral entrapment of the cauda equina within the elements of the fractured lamina. A dural tear is likely in this scenario as well. It is recommended that this type of burst fracture be treated surgically with laminectomy, freeing of the entrapped nerve roots, and dural repair followed by stabilization of the fracture by either a posterior or combined approach. The degree of kyphosis and the extent of anterior canal compromise does not warrant corpectomy in this patient. Therefore, after completing the laminectomy and dural repair, posterior fusion and instrumentation should be sufficient to stabilize the fracture. Cammisa FP Jr, Eismont FJ, Green BA: Dural laceration occurring with burst fractures and associated laminar fractures. J Bone Joint Surg Am 1989;71:1044-1052.

The MRI scans show Modic changes in the L4-L5 vertebral bodies due to spondylosis. There is no increased fluid signal or enhancement in the L4-L5 disk to suggest infection or any other pathologic process. Therefore, the patient's pain should be treated with a course of physical therapy and rehabilitation. There is no infection; therefore, IV antibiotics and debridement are not indicated. Similarly, a pseudomeningocele is not present. A revision diskectomy is useful for recurrent radiculopathy but would not be helpful for degenerative low back pain. Spivak JM, Connolly PJ (eds): Orthopaedic Knowledge Update: Spine 3. Rosemont, IL, American Academy of Orthopaedic Surgeons, 2006, pp 319-329.

Cotler and associates reported on the use of awake skeletal traction to reduce facet fracture-dislocations in 24 patients. Seventeen patients required more than 50 pounds of traction (the "traditional" limit) to achieve reduction. More than 100 pounds of traction was safely used in one-third of the patients in this study. A cadaver study has supported the safe use of traction with weights in excess of 100 pounds. Cotler JM, Herbison GJ, Nasuti JF, et al: Closed reduction of traumatic cervical spine dislocation using traction weights up to 140 pounds. Spine 1993;18:386-390.

Although the imaging reveals generalized lumbar spondylosis and stenosis, in particular at L4-5, the MRI scan of the left hip clearly reveals a stress fracture of the femoral neck. Therefore, the treatment of choice is non-weight-bearing of the left lower extremity. During the evaluation of acute back pain, clinicians must include other possibilities within the differential diagnosis that may mimic mechanical axial back pain; thus, potential complications from a missed diagnosis can be avoided. Wong DA, Transfeldt E: Macnab's Backache, ed 4. Philadelphia, PA, Lippincott Williams and Wilkins, 2007, pp 339-361.

The patient has symptoms that are more consistent with vascular claudication than with the pseudoclaudication anticipated from lumbar spinal stenosis. Therefore, the patient is a candidate for further vascular work-up. The radiographs reveal early spinal stenosis and spondylolisthesis at L4-5 but also show significant calcification of the iliac arteries, suggestive of peripheral vascular disease. Vascular claudication is a manifestation of peripheral vascular disease and presents with crampy leg pain that is exacerbated by physical exertion. The pain is easily relieved by standing still or sitting. Unlike pseudoclaudication, a forward-flexed posture and/or sitting does not improve the symptoms. Night pain is common in vascular claudication due to the elevation of the extremities and patients often report pain improvement by hanging their extremities in a dependent position. In evaluation of a patient with suspected vascular claudication, the five "P's" of vascular insufficiency should be monitored, including pulselessness, paralysis, paresthesia, pallor, and pain. While pain and paresthesias can be common in both vascular claudication and pseudoclaudication, the presence of any of the remaining symptoms is suggestive of vascular disease. Aufderheide TP: Peripheral arteriovascular disease, in Rosen P, Barkin R (eds): Emergency Medicine: Concepts and Clinical Practice, ed 4. St Louis, MO, Mosby, 1998, pp 1826-1844.

Tuberculosis of the spine is seen in 50% to 60% of skeletal disease and is most commonly found in the lower thoracic or upper lumbar spine. Typically two or more adjacent bodies are involved as seen in this MRI scan. The disk space is narrowed but still relatively preserved as opposed to pyogenic infections (black arrow). Epidural extensions often spread from vertebrae to vertebrae (white arrow); however, the posterior elements are not frequently involved (arrowhead). Tumors rarely spread to adjacent vertebrae. The anterior and posterior spread of the infectious process rules out trauma. Boachie-Adjei O, Squillante RG: Tuberculosis of the spine. Orthop Clin North Am 1996;27:95-103.

With the notable exception of fusion for degenerative spondylolisthesis and scoliosis, there is a paucity of evidence on the indications for spinal fusion in patients undergoing laminectomy for spinal stenosis. However, it is generally recommended that if the spine is destabilized (for example by removal of one complete facet joint or by an iatrogenic pars fracture), spinal fusion should be considered. Although fusion can be considered for a very long laminectomy, a two-level laminectomy does not represent, by itself, a clear indication for the addition of a spinal fusion. The repair of a dural tear and the use of nicotine by the patient play no role in the determination of whether or not to add fusion to a laminectomy procedure. Spivak JM, Connolly PJ (eds): Orthopaedic Knowledge Update: Spine 3. Rosemont, IL, American Academy of Orthopaedic Surgeons, 2006, pp 299-409.

The sagittal and axial CT scans show a bilateral spondylolysis at L5. The defect is in the pars interarticularis on the right side but at the base of the pedicle on the left. Having failed a trial of physical therapy with only a 3-month history of pain, the next most appropriate step in management should consist of activity modification and bracing in an antilordotic lumbosacral orthosis. Surgical intervention is reserved for patients who have failed to respond to a trial of bracing and activity restriction. Debnath UK, Freeman BJ, Grevitt MP, et al: Clinical outcome of symptomatic unilateral stress injuries of the lumbar pars interarticularis. Spine 2007;32:995-1000.

Although symptomatic thoracic disk herniations can affect more caudal structures, even to the point of paralysis, the pattern of radiating pain has been described as either following the dermatomal band around the chest or feeling to the patient as if the pain passes straight anteriorly to the chest wall.

The patient has pain involving the cervical and lumbar spine as well as pain and swelling in both the knees and ankles. As such, this can be classified as polyarticular arthritis. The presence of multiple joint symptoms in the lower extremities, the absence of a history of trauma, and the multiple joints involved direct attention away from the spine as the etiology of this patient's pain. Radiographs of the involved joints are not likely to yield much useful information to assist with a diagnosis. Likewise, an MRI scan of the lumbar spine is not likely to provide much information regarding the etiology of the patient's condition. When a rheumatologic illness is suspected, the selective use of confirmatory laboratory testing can aid in arriving at a correct diagnosis. A presumed case of gout or chondrocalcinosis can be confirmed by the presence of the appropriate crystals in a joint-fluid aspiration. Because of the patient's recent trip to Costa Rica and the subsequent gastroenteritis, a CBC count, ESR, and CRP should be ordered to rule out infectious and inflammatory versus noninflammatory conditions. Rheumatoid factor (RF) in general should only be ordered for patients with polyarticular joint inflammation for more than 6 weeks. The presence of rheumatoid factor does not indicate rheumatoid arthritis. Antinuclear antibodies (ANA) should be ordered when a connective tissue disease such as systemic lupus erythematosus (SLE) is suspected on the basis of specific history and physical examination findings, such as inflammatory arthritis. Human leukocyte antigen-B27 (HLA-B27) should be ordered only when the patient's history is compatible with ankylosing spondylitis or Reiter's syndrome and this patient had a history of Reiter's syndrome. Gardner GC, Kadel NJ: Ordering and interpreting rheumatologic laboratory tests. J Am Acad Orthop Surg 2003;11:60-67.

Atlas and associates, in the Maine Lumbar Spine Study, reported that overall, patients treated initially with surgery reported better outcomes. By 5 years, 19% of surgical patients had undergone at least one additional lumbar spine operation, and 16% of nonsurgical patients had opted for at least one lumbar spine operation. At the 5-year follow-up, 70% of patients initially treated surgically reported improvement in their predominant symptom (back or leg pain) versus 56% of those initially treated nonsurgically. They also noted that there was no difference in the proportion of patients receiving disability compensation at the 5-year follow-up.

The injury shown is a fracture-dislocation and it is highly unstable. In addition to this concern, spinal epidural hematomas have a much higher incidence in people with ankylosing spondylitis following knee fracture. It is felt to be due to disrupted epidural veins, with hypervascular epidural soft tissue in the setting of a rigid spinal canal. Patients with ankylosing spondylitis may have other significant comorbidities, especially cardiac and pulmonary, and these should be carefully assessed. Ludwig S, Zarro CM: Complications encountered in the management of patients with ankylosing spondylitis, in Vaccaro AR, Regan JJ, Crawford AH, et al (eds): Complications of Pediatric and Adult Spine Surgery. New York, NY, Marcel Dekker, 2004, pp 279-290.

Retrograde ejaculation is the sequela of an injury to the superior hypogastric plexus. This structure needs protection, especially during anterior exposure of the lumbosacral junction. Although the superior hypogastric plexus can be injured with anterior or anterolateral spine surgery at any lumbar level, it is most at risk with anterior transperitoneal approaches to the lumbosacral junction. To avoid this complication, the use of monopolar electrocautery should be avoided during deep dissection in this region. The ideal anterior exposure starts with blunt dissection just to the medial aspect of the left common iliac vein sweeping the prevertebral tissues toward the patient's right side. Flynn JC, Price CT: Sexual complications of anterior fusion of the lumbar spine. Spine 1984;9:489-492. Watkins RG (ed): Surgical Approaches to the Spine. New York, NY, Springer-Verlag, 1983, p 107.

The hypoglossal nerve exits from the ansa cervicalis at approximately the C2-3 level and can be injured during retraction up to the C2 level. The superior laryngeal nerve lies at about C4-5. The facial nerve is much higher. The vagus nerve runs with the internal jugular and carotid much more laterally. The phrenic nerve exits posteriorly. Chang U, Lee MC, Kim DH: Anterior approach to the midcervical spine, in Kim DH, Henn JS, Vaccaro AR, et al (eds): Surgical Anatomy and Techniques to the Spine. Philadelphia, PA, Saunders Elsevier, 2006, pp 45-54.

The injury pattern is that of a burst fracture at L1 contiguous with a compression fracture at T12. There is associated kyphosis and slight spondylolisthesis of T12 on L1. Treatment of this type of burst fracture in neurologically intact patients is somewhat controversial, with at least one study demonstrating equal long-term results comparing nonsurgical treatment to surgical treatment. In this study, however, body casts were used initially in the nonsurgical group. Moreover, because this patient has multiple fractures, spinal fracture stabilization should be considered to facilitate early mobilization. Surgical stabilization and fusion via a posterior approach is the best treatment option in this patient. Anterior decompression is not necessary since the patient is neurologically intact. McLain RF, Benson DR: Urgent surgical stabilization of spinal fractures in polytrauma patients. Spine 1999;24:1646-1654. Wood K, Butterman G, Mehbod A, et al: Operative compared with nonoperative treatment of a thoracolumbar burst fracture without neurological deficit: A prospective, randomized study. J Bone Joint Surg Am 2003;85:773-781.

Incomplete cord syndromes have variable neurologic findings with partial loss of sensory and/or motor function below the level of injury. Incomplete cord syndromes include the anterior cord syndrome, the Brown-Séquard syndrome, central cord syndrome, and posterior cord syndrome. Central cord syndrome is characterized with greater motor weakness in the upper extremities than in the lower extremities. The pattern of motor weakness shows greater distal involvement in the affected extremity than proximal muscle weakness. Anterior cord syndrome involves a variable loss of motor function and pain and/or temperature sensation, with preservation of proprioception. The Brown-Séquard syndrome involves a relatively greater ipsilateral loss of proprioception and motor function, with contralateral loss of pain and temperature sensation. Posterior cord syndrome is a rare injury and is characterized by preservation of motor function, sense of pain, and light touch, with loss of proprioception and temperature sensation below the level of the lesion. Spinal shock is the period of time, usually 24 hours, after a spinal injury that is characterized by absent reflexes, flaccidity, and loss of sensation below the level of the injury. Penrod LE, Hegde SK, Ditunno JF: Age effect on prognosis for functional recovery in acute, traumatic central cord syndrome. Arch Phys Med Rehab 1990;71:963-968.

Kayanja and associates, in a number of biomechanical studies, showed that in a kyphotic spine the strain is located at the apex of the deformity, the force is transmitted to the superior adjacent vertebrae, and that realignment and cement augmentation effectively normalize the load transfer. Kayanja MM, Ferrara LA, Lieberman IH: Distribution of anterior cortical shear strain after a thoracic wedge compression fracture. Spine J 2004;4:76-87. Kayanja MM, Togawa D, Lieberman IH: Biomechanical changes after the augmentation of experimental osteoporotic vertebral compression fractures in the cadaveric thoracic spine. Spine J 2005;5:55-63. Kayanja MM, Schlenk R, Togawa D, et al: The biomechanics of 1, 2, and 3 levels of vertebral augmentation with polymethylmethacrylate in multilevel spinal segments. Spine 2006;31:769-774.